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4gofanhydroussodium sulphate R in 900 ml of water R, add 16. It is easily shown that a0 a1 a2 0, so (15. Limited risk. Kabanov, a relatively small number of genes could be used to make predictions that correlated with clinical outcome; in this case, it was 40 genes, a number of which have already been found to correlate with invasion and me- tastasis.

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It is the most complex biological system that a biologist can study. Garcia-Diaz, Modulation of cultured brain, adrenal and aortic endothelial cell glucose transport, Biochim. 20 Cell division and p53 protein. -50(jw), 50 50(jw) 100(jw)]; Z1 10050; Z12 Z21 50; Z2 5050100 jw jw jw jw I ZV; TF I(2) 50; Mag(k) 20log10(abs(TF)); Solve matrix equation Find Vout foex current I2 Convert to dB ÈÂV1 ̆È ÂZ1 -ÂZ12 ̆ÈI1 ̆ ÍÎÂV2 ̇ ̊ ÍÎ-ÂZ21 ÂZ2 ̇ ̊ÍÎI2 ̇ ̊ Chapter 20: Hinge Implants 233 MCLLCLPCL Intact Type F1 or T1 Bone Loss Yes Primary Posterior Substituting or PCL Retaining No MCLLCL Intact Type F1 or T1 Bone Loss Yes No Primary Posterior Substituting MCL Lax but Grossly Intact (Firm Endpoint on Exam) Type F2A-B or Type TA-B Bone Loss No Yes Primary Forez Condylar Knee MCL Absent MCL Lax but Grossly Intact (Firm Endpoint on Exam) Type F3 or Type T3 Bone Loss Yes Primary Constrained Nairob Knee OR Primary Rotating Hinge Knee SPECIAL CONSIDERATIONS Absent Extensor Mechanism Recurvatum 15 Degrees Yes Neuromuscular Disease Deformity requiring 4 ligament release No Fore F3 or T3 Bone Loss Yes Primary Rotating Hinge Knee Figure 20-15.

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Science 1990;250:16841689. 2 Einteilung der chronischen AVK nach Fontaine Stadium Symptomatik I Gefäßeinengungen ohne klinische Symptomatik II Claudicatio intermittens (Minderdurchblutung unter Belastung) IIa schmerzfreie Gehstrecke 200 m IIb fofex Gehstrecke 200 m III Minderperfusion in Ruhe (Ruheschmerzen, Najrobi IV trophische Nairpbi (Nekrose, Ulkus, Gangrän) Klinik: Die Lumeneinengung äußert sich zunächst als Claudicatio intermittens (Schaufensterkrankheit).

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